Various video postings from alternavita on FB 

Not in any particular order but a brief description in left column.


Progurt for Progress and leaving Facebook. 


I’ve done extensive research on anti parasitics. This video explains why I believe Ivermectin is effective for the ‘Golden Bug’. And my commentary on the ‘hoax’ that doesn’t exist but essentially is the same risk groups as seen in history.

It’s laughable to isolate a virus from stool samples considering the average 5 grams of any stool sample contains over 10,000 viruses. Virus reservoirs are in the intestinal tract.

This official story is kind of like the FBI showing up to solve a crime they just committed.

Asians and over 10% of world populations use ‘night soil’ which is untreated human waste to fertilize crops. In high outbreak cities in the US sanitation is extremely poor.

In addition to poor sanitation I’m also guessing as with SARS and MERS and AIDS  ‘sex playground’ travel destination/s was a key factor. I’m also guessing use of poppers or other drugs may be responsible for the high number of associated pneumonia initial presentations. 

For ANY infectious or parasitic disease to start, it is ALWAYS a requisite that the host suffer IMMUNODEFICIENCY. At the same time, infectious and parasitic diseases themselves cause additional IMMUNE SUPPRESSION and more MALNUTRITION. This immune suppression is SECONDARY to the accumulation of free radicals, especially oxidizing species, that occurs during and after infectious and parasitic diseases.

Clinical Aspects of Immunology and Biochem J.


A brief overview of my favorite probiotics. I meant to say you don’t need enzymes, that is what your healthy intestine (enterocytes) are for.


Why oleic acid?

Most of the fatty acids bound to human and bovine DBP are monounsaturated and saturated, mainly oleic and palmitic acids, which together account for 50% of the total of fatty acids in both species. By contrast, polyunsaturated fatty acids represented a minor component, less than 5%.

Vitamin D Resistance

Vitamin 1,25-D3 inhibits proliferation of T helper 1(Th1) cells consequently impairing production of interferon (IFN)], as well as T helper 17 skewing cytokine production toward (Th2)phenotype…..Vitamin D-induced down regulation of the cytokine response may not always be associated with optimal host defense. The Th1 cytokine profile is vital for clearance of certain organisms and ancillary immune activity. Effective clearance will depend on appropriate macrophage activation. The presence of 1,25- D3 disrupts this pathway, as IFN≥ secretion is blocked, impairing macrophage activation.
Persistent blockade of these inhibitory receptors has lead to the breakdown in immune self tolerance, thereby increasing susceptibility to autoimmune or auto-inflammatory side effects, including rash, colitis, hepatitis and various endocrinopathies.
Resistance to adrenal and gonadal steroids as well as the vitamin D hormone, 1,25- dihydroxyvitamin D (1,25-(OH) 2 D). DBP binds vitamin D metabolites in serum and act as a carrier for these metabolites preventing cells from a massive uptake and its toxic effects.
Vitamin D resistance  also correlates with high circulating levels of other steroid hormones including glucocorticoid. (mineral corticoid, progesterone, testosterone, 17 b-estradiol).
Resistant species are not clinically affected as long as there is adequate substrate available for accelerated steroid/sterol hormone synthesis.
That is oleic acid and sulfate.
Tolerance is the ability of the immune system to ‘see’.

Both forms of synthetic Vitamin D are poisons. Vitamin D3 is not recommended in natural oral immune therapy. I only recommend cod liver oil


The Golden Bug, the truth about PCR and the perpetual fraud regarding ‘infections’ as perpetrated by Anthony Fauci and DS medical system.

I often hear that PCR test is a scam, it is NOT a scam; it is very effective for its intended purpose which originally was to screen blood to ensure safety profiles and which is why the inventor Kary Mullis won the Nobel Prize.

Breastfeeding, food and water security are major protective factors against malnutrition and critical factors in the maturation of healthy gut microbiota, characterized by a transient bifidobacterial bloom before a global rise in anaerobes. Early depletion in gut Bifidobacterium longum, a typical maternal probiotic, known to inhibit pathogens, represents the first step in gut microbiota alteration associated with severe acute malnutrition (SAM). Later, the absence of the Healthy Mature Anaerobic Gut Microbiota (HMAGM) leads to deficient energy harvest, vitamin biosynthesis and immune protection, and is associated with diarrhea, malabsorption and systemic invasion by microbial pathogens. A therapeutic diet and infection treatment may be unable to restore bifidobacteria and HMAGM. Besides re-feeding and antibiotics, future trials including non-toxic missing microbes and nutrients necessary to restore bifidobacteria and HMAGM, including prebiotics and antioxidants, are warranted in children with severe or refractory disease.
Tolerance is the ability of the immune system to ‘see’.
Clinically chronic inflammatory disorder of the alimentary tract leads to failure to rebuild immune memory.

Endotoxemia is a critical component in the

development of hemolytic uremic syndrome in

a mouse model that closely mimics the condition

in humans. Since this condition is caused

by the ingestion of E. coli strains that express

both a toxin (Shiga-like toxin 2) and LPS,

oral administration of colostrum will treat

and ameliorate the disease. Bovine colostrum

ameliorates diarrhea in infection with diarrheogenic

E. coli, Shiga toxin-producing E. coli

and E. coli expressing intimin and hemolysin it

supports wound healing and also the regeneration

of damaged intestinal mucosa.

Inflammatory disorders of the alimentary tract may involve any one of more of the various tissues, regions, structures or organs of the alimentary tract including the mucosa (mucositis), mouth (stomatitis), tongue (glossitis),  gums (gingivitis), oesophagis (oesophagitis), stomach (gastritis), colon (colitis), ilieum (ileitis), liver (hepatitis), gallbladder (cholecystitis), pancreas (pancreatitis), or parotid salivary gland (parotitis). Clinically, the inflammatory disorder of the alimentary tract may present as increased permeability leading to diarrhea, enteropathy, pain, bloating, constipation, anorexia or malabsorption leading to a decline in body weight and failure to rebuild immune memory in the alimentary tract, hypergammaglobulinaemia and cachexia.
IBD is not just leaky gut
* Intestinal barrier loss alone is insufficient to initiate disease (IBD).