Recipe/How To©: How To Detox The Liver


How To Detox The Liver

Use healthy fat to move out fat, increase autophagy and decrease toxin load with specific probiotics

Treating SRB’s with probiotics genus Bifidobacterium species.

Components of the human microflora were found to inhibit the growth of SRB and their H2S production efficiently. Anaerobic viable bacteria such as bifidobacteria, sulfate reduction of sulfate-reducing bacteria can be inhibited by inhibiting the growth of these bacteria. Due to the strong infiltration with macrophages at the site of inflammation and the additional exposure to NO and hydrogen peroxide is formed an uncontrolled reaction mixture. This has the consequence that H2S is chemically re-oxidized to thiosulfate or even sulfite. Both compounds are reduced by SRB again to H2S, so that a vicious circle is created with low available sulfate. Much of the SRB reaches certainly over the manure fertilization in the food chain. In addition, infants who are not breastfed are at an early stage with SRB (and with methane-forming bacteria) causing a settled condition.

Other Liver Helpers

Other liver helpers include choline, saturated fat, healthy oils including cod liver oil, and autophagy or pattern dieting, which is a daily fast of at least 13 hours.

Low fat diets do not aid liver detoxification. Excess water intake is not especially helpful as it flushes out minerals. Fruit juices with the possible exception of citrus are the last thing that are helpful for liver detox due to the fructose content. Apple cider vinegar with live bacteria can increase autophagy. A high lean protein keto diet is often not helpful in patients with advanced liver or kidney failure due to toxic metabolites albeit malnutrition is often acute in these patients.

The amino acids aspartic and glutamic acid can detoxify ammonia by amidation. But this therapy with amino acids is best used only in the early stages of liver damage. To reduce the formation of ammonia in liver damage some amino acids are broken down to ammonia, and increase the ammonia levels in plasma.


The influence of amino acids and proteins is always a balancing act between the supply of nitrogen while trying to avoid the potential increase of the ammonia, and the need to provide patients with essential amino acids. Patients are usually dependent on a high-calorie diet, which should include proteins. Cirrhotic patients are therefore particularly fed protein-rich diets as long as possible. However, since the protein tolerance is reduced increasingly in these patients, this in turn leads to an increase in ammonia levels in the plasma. The inclusion of normal milk can increase in blood ammonia level in healthy subjects, and in cirrhosis patients.

More surprising was the finding that one can achieve with colostrum or with the whey from this colostrum that the blood ammonia level is reduced dramatically in patients with liver cirrhosis.

*Over time as the gut and liver heals whole fat mammal milk is not a problem and lactose/galactose is a necessary sugar for immune function, except in galactosemia.

Immune Health News

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Malnutrition  is the leading cause of death worldwide in children under the age of five, and is the focus of the first World Health Organization (WHO) Millennium Development Goal. Breastfeeding, food and water security are major protective factors against malnutrition and critical factors in the maturation of healthy gut microbiota, characterized by a transient bifidobacterial bloom before a global rise in anaerobes. Early depletion in gut Bifidobacterium longum, a typical maternal probiotic, known to inhibit pathogens, represents the first step in gut microbiota alteration associated with severe acute malnutrition (SAM). Later, the absence of the Healthy Mature Anaerobic Gut Microbiota (HMAGM) leads to deficient energy harvest, vitamin biosynthesis and immune protection, and is associated with diarrhea, malabsorption and systemic invasion by microbial pathogens. A therapeutic diet and infection treatment may be unable to restore bifidobacteria and HMAGM. Besides re-feeding and antibiotics, future trials including non-toxic missing microbes and nutrients necessary to restore bifidobacteria and HMAGM, including prebiotics and antioxidants, are warranted in children with severe or refractory disease.