How To Detox The Liver
Use healthy fat to move out fat, increase autophagy and decrease toxin load with speciﬁc probiotics
Treating SRB’s with probiotics genus Biﬁdobacterium species.
Components of the human microﬂora were found to inhibit the growth of SRB and their H2S production eﬃciently. Anaerobic viable bacteria such as biﬁdobacteria, sulfate reduction of sulfate-reducing bacteria can be inhibited by inhibiting the growth of these bacteria. Due to the strong inﬁltration with macrophages at the site of inﬂammation and the additional exposure to NO and hydrogen peroxide is formed an uncontrolled reaction mixture. This has the consequence that H2S is chemically re-oxidized to thiosulfate or even sulﬁte. Both compounds are reduced by SRB again to H2S, so that a vicious circle is created with low available sulfate. Much of the SRB reaches certainly over the manure fertilization in the food chain. In addition, infants who are not breastfed are at an early stage with SRB (and with methane-forming bacteria) causing a settled condition.
Other Liver Helpers
Other liver helpers include choline, saturated fat, healthy oils including cod liver oil, and autophagy or pattern dieting, which is a daily fast of at least 13 hours.
Low fat diets do not aid liver detoxiﬁcation. Excess water intake is not especially helpful as it ﬂushes out minerals. Fruit juices with the possible exception of citrus are the last thing that are helpful for liver detox due to the fructose content. Apple cider vinegar with live bacteria can increase autophagy. A high lean protein keto diet is often not helpful in patients with advanced liver or kidney failure due to toxic metabolites albeit malnutrition is often acute in these patients.
The amino acids aspartic and glutamic acid can detoxify ammonia by amidation. But this therapy with amino acids is best used only in the early stages of liver damage. To reduce the formation of ammonia in liver damage some amino acids are broken down to ammonia, and increase the ammonia levels in plasma.
The inﬂuence of amino acids and proteins is always a balancing act between the supply of nitrogen while trying to avoid the potential increase of the ammonia, and the need to provide patients with essential amino acids. Patients are usually dependent on a high-calorie diet, which should include proteins. Cirrhotic patients are therefore particularly fed protein-rich diets as long as possible. However, since the protein tolerance is reduced increasingly in these patients, this in turn leads to an increase in ammonia levels in the plasma. The inclusion of normal milk can increase in blood ammonia level in healthy subjects, and in cirrhosis patients.
More surprising was the ﬁnding that one can achieve with colostrum or with the whey from this colostrum that the blood ammonia level is reduced dramatically in patients with liver cirrhosis.