February, 5 2019©
The greatest charity one can do to another is to lead him to the truth.
St. Thomas Aquinas
In the US alone 75 million people are high risk for NAFLD.
The global prevalence of NAFLD is currently estimated to be 24%.
NAFLD is one of the most important causes of liver disease worldwide and will probably emerge as the leading cause of end-stage liver disease in the coming decades, with the disease affecting both adults and children. The epidemiology and demographic characteristics of NAFLD vary worldwide, usually parallel to the prevalence of obesity, but a substantial proportion of patients are lean. The large number of patients with NAFLD with potential for progressive liver disease creates challenges for screening, as the diagnosis of NASH necessitates invasive liver biopsy. Furthermore, individuals with NAFLD have a high frequency of metabolic comorbidities and could place a growing strain on health-care systems from their need for management. While awaiting the development effective therapies, this disease warrants the attention of primary care physicians, specialists and health policy makers.
That is also a high risk for immune deficiency, and the majority of those people have liver problems long before they get diagnosed with acute liver failure.
A dysfunctional autophagic mechanism leads to chronic intestinal inﬂammation in IBD.
The pathogenesis of the liver manifestation of IBD is related to gut inﬂammation that results in inﬂamed portal tracts of the enterohepatic circulation of lymphocytes from the gut to the liver.
The vitamin D binding protein (DBP) was initially discovered as a major liver-derived polymorphic protein and called group-specific component or Gc.
Vitamin D Binding Protein is otherwise know as:VDBP Gc-Protein GlycoproteinTransport protein Gc-Globulin (GcMAF is Gc globulin macrophage activating factor.
Serum levels of Gc-globulin are often markedly reduced and the reduction correlates with the severity and the acuity of liver disease, since Gc-globulin concentrations were lowest among patients with ALF (acute liver failure), a condition characterized by massive hepatic necrosis.
• Patients with ALF often develop
- renal failure
- arterial hypotension
- severe infections
- and occasionally pulmonary dysfunction
The most feared complication in ALF is the development of cerebral edema and intracranial hypertension.
Liver failure is also associated with MOD (multi organ dysfunction).
In general, total Gc-globulin levels are decreased in all patients with hepatotoxicity. Patients with ALF have total Gc-globulin concentrations of approximately 100 mg/L which is less than one third of normal values.
You can get a clinical Gc globulin test to determine your levels of Gc globulin if you feel you are at risk for low levels.
The downstream effects of alcohol
Anti oxidants are best supplied by food, not supplements. Buttermilk is a superior anti oxidant.
The oils for liver failure include:
Daily intake of these 4 oils combined equals about 1 tbsp.
- Cod liver oil (1/2-1 tsp. or 1 soft gel)(winter only, switch to fish oil summer)
- Olive oil (1 tsp.)
- Grass fed butter (1 tsp.)
Immune Health News